Thus we expect the edge temperatures to be greater than those shown in the graphs.
Its resolution allows to eliminate locally the edge temperatures as function of the mean temperature in each cell.
Average cell temperature conditions of 15, 25, and 35 °C with edge-to-edge temperature differences (ΔT) of 0, 5, 10, and 20 °C are applied.
For each cell, the two half-edge normal fluxes attached to a node are expressed in terms of the half-edge temperatures impinging at this node and the cell-centered temperature.
The space discretization relies on the introduction of two half-edge normal fluxes and two half-edge temperatures per cell interface using the partition of each cell into sub-cells.
The elimination of the half-edge temperatures is performed locally at each node by solving a small linear system which is obtained by enforcing the continuity condition of the normal heat flux across sub-cell interface impinging at the node.
As indicated by the directed edge between temperature and asthma exacerbations, temperature may have direct effects on inflammation pathways or airway hyper-responsiveness causing asthma exacerbations.
The data include wound measurement variables, for example, wound edge distance, temperature, include exudate amount, etc.
The influences of the volume fraction index, edge compression, temperature change, boundary condition, side-to-thickness ratio and plate aspect ratio are also discussed.
These indirect effects are represented in the causal graph by the directed edge between temperature and asthma triggers, and between asthma triggers and asthma exacerbations.
