Sentence examples for edema ratio from inspiring English sources

Examinations included a STIR (short tau inversion recovery) sequence for calculation of edema ratio (ER), a T1-weighted spin-echo sequence for assessment of global relative enhancement (gRE), and inversion-recovery sequences to visualize late gadolinium enhancement (LGE).

The T2-weighted images for the calculation of the edema ratio (ER) and the T1-weighted spin-echo images to calculate the global relative enhancement (gRE) were acquired using the body coil to avoid influence of signal heterogeneities on signal intensity measurements as previously described [ 14, 15].

One SNP, rs6123045, in the nuclear factor of activated T-cells cytoplasmic calcineurin-dependent 2 (NFATC2) gene was significantly associated with edema (odds ratio [OR] 1.89 [95% CI 1.47 2.42]; P = 5.32 × 10−7, corrected P = 0.017) (Fig. 1 A).

Diagnostic criteria for ALI/ARDS included all of the following: acute bilateral pulmonary infiltrates on a chest x-ray consistent with the presence of pulmonary edema, a ratio of arterial oxygen tension to fraction of inspired oxygen (PaO2/FiO2) of 100 to 300 mm Hg for ALI/ARDS, and the clinical absence of left atrial hypertension.

Homozygous individuals had the highest edema rate (hazard ratio 2.89, P = 4.22 × 10−4) when compared with individuals homozygous for the protective allele, with heterozygous individuals having an intermediate risk.

Significant increases in risk of TMJ pain occurred with disk displacement without reduction in combination with osteoarthrosis and bone marrow edema (3.7 1 odds ratio; P =.000) and with disk displacement without reduction in combination with osteoarthrosis and effusion (2.8 1 odds ratio; P =.000).

Also, we performed the same matching analysis without patient with ARF related to cardiogenic edema and odd ratio was 1.88 (0.71–5.00), p = 0.50.

In this study, we quantified the peritumoral edema as a ratio to tumor size.

Immediately after induction of alveolar edema, the PaO2/FiO2 ratio sharply decreased in all groups except in the SE + PEEP group.

Our in vitro investigations were supported by in vivo studies which indicate that the nonselective opioid agonist, biphalin, achieves enhanced neuroprotective potency compared to any of the selective opioid agonists, evidenced by reduced edema and infarct ratios.

17 patients had lung injury with radiological evidence of pulmonary edema and/or a paO2/FiO2 ratio of <300 on admission.