Sentence examples for ec to induce from inspiring English sources

CXCL12 can act directly on EC to induce angiogenesis (reviewed in Salcedo and Oppenheim).

EC associated disease outbreaks were also reported from ducks in Germany [ 18, 19] and EC infection was successfully reproduced experimentally in Pekin ducklings [ 20], demonstrating the ability of EC to induce disease in other birds than chickens.

It is likely that Rnd2 and Rnd3 act through a similar mechanism in EC to induce actomyosin contractility, but Rnd2 induces the strongest response, perhaps because it cannot effectively interact with p190RhoGAP and downregulate RhoA activity, whereas Rnd1 and Rnd3 can both act through this pathway (Wennerberg et al., 2003).

One strategy included the delivery of polymers that produce or release NO, a compound released by ECs to induce vasorelaxation and minimize SMC migration, both of which suppress intimal hyperplasia.

Recognition of cell surface ATP synthase at low pHe microenvironment contributes to intracellular acidification of EC known to induce cell death and the triggering of inflammation.

Corroborating the specificity of the cellular responses, the reverse sequence A β40-1 peptine, in difference to A β-WT, was unable to trigger EC apoptosis or to induce activation of caspase-8 and caspase-9 under the conditions tested (Supplementary Figure 2).

To induce EC apoptosis, ECs were pre-treated 1 h with 100 µM pyrrolidine dithiocarbamate (PDTC, Sigma Aldrich, St . Louis MO, USA) before TNF addition and incubation for 24 h.

DOI: http://dx.doi.org/10.7554/eLife.00999.012 Interestingly, when Yki was expressed in ECs using Myo1A-Gal4 to induce non-autonomous ISC proliferation, the number of ISCs/EBs was increased with high levels of Brm in the nucleus (compare Figure 5P P′′′ with Figure 5O O′′′), suggesting that Yki-induced non-autonomous ISC proliferation also induces high levels of Brm in nuclei of ISCs and EBs.

VEGF, an EC-specific angiogenic cytokine, has been shown to induce EC proliferation and migration, increase vascular permeability, stimulate disassociation of pericytes from vascular endothelium (Morikawa et al, 2002) and act as a key survival factor for EC (Gerber et al, 1998).

Immobilized RGDS facilitated initial EC adhesion on the surfaces and INS modification was demonstrated to induce EC proliferation, respectively.

The synthesis is a facile bulk calcination in which the inexpensive polymer ethyl cellulose (EC) is used as a structure-directing agent; the EC concentration is controlled to induce spherical morphology.