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Thus, early parasites had a shorter latency time.
If anything, early parasites produced more, rather than fewer, reproductive forms than did late parasites.
Early parasites were also more virulent, in agreement with theoretical models predicting that higher virulence evolves under increased background mortality [ 11, 15].
Further analyses suggested that thousands of genes were lost as an ancient free-living algae evolved into the apicomplexan ancestor, and further losses occurred as these early parasites evolved into modern species.
First, the mean density of reproductive forms of early parasites was only marginally larger than that of late parasites after the first week, and this difference was not statistically significant.
The negative relationship between the densities of reproductive forms after the first week and latency time indicates such a link in our experiment, but there was no clear-cut evidence for faster within-host replication of early parasites.
Similar(51)
These macrophage-inhibitory cytokines may contribute to the establishment of the infection and early parasite replication.
Activated macrophages are involved in early parasite killing [10].
Mice lacking B or T cells exhibit early parasite dynamics that are very similar to those in wild type mice [6] [9].
Sequence characteristics of the five 454 normalized libraries derived from skeletal muscle, intestine, blood and head kidney (HK-38, early parasite infection; HK 105, chronic parasite infection).
When S. griseus chitinase was included with their infections they found that the early parasite mortality was exacerbated, corresponding to the lack of an effective PM barrier.
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