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Although these regulators act to promote stable myosin accumulation and apical cell constriction, loss-of-function phenotypes for each of these pathway members is not equivalent, suggesting the existence of additional ventral furrow regulators.
However to date the distinct roles played by each of these pathway metabolites in mediating direct- and indirect-defense reponses has remained elusive.
For each of these pathway networks an optimally arranged 2D grid representation was produced as described in Results.
We then used an SVM with features P j in discriminating the classes in the training set, and obtained SVM weights W j for each of these pathway features (obtained from combined data across the training samples k).
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For each of these pathways, 3 seed genes were chosen at random.
Each of these pathways can impair endothelial function, a unifying aspect of the pathophysiology of sepsis.
Third, calculating the mass balance for each of these pathways we deduce the gross negative emissions needed to be compatible with the RCP2.6 scenario.
The paper examines the evidence from qualitative as well as quantitative studies on each of these pathways.
The identification of distinct molecular requirements for each of these pathways makes it clear that mammalian cells have evolved more complex targeting mechanisms and routes than previously anticipated.
The regulatory approach involves, primarily, two major sets of regulations; and the scientific basis for product categorization and the application of each of these pathways is discussed.
For each of these pathways except PFL, we see examples where ethanol production has been engineered with a yield of >90% of the theoretical maximum.
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CEO of Professional Science Editing for Scientists @ prosciediting.com