Understanding on distinct subsets of endothelial progenitor cells may provide insights of endothelial dysfunction or repair in the acute ischemic event.
We also have strong interests in development, disease, dysfunction, and repair of the nervous system, and in connecting basic research to translational medicine.
D.S. is supported by a Scholar Award of the James S. McDonnell Foundation (Function, dysfunction and repair of language networks).
Indeed, while older age at onset is associated with a more rapid disability progression likely due to prevalence of the primary progressive disease course, age-dependent degenerative processes, and dysfunction of repair mechanisms in older subjects early onset MS patients reach disability milestones at a younger age compared to late onset MS cases, even though in a longer time interval [ 26, 75].
Importantly, this article broadens the perspective of research into measuring risk of dementia from focusing on specific neuropathological markers of dementia subtypes, to mechanisms underlying more general bodily vitality and health, as well as dysfunctions in repair.
Importantly, Song and colleagues [ 1] broaden the perspective of research into measuring risk of dementia from focusing on specific causes of neuropathological markers of dementia subtypes, to mechanisms underlying more general bodily vitality and health, as well as dysfunctions in repair.
In the Synthetic Neurobiology Group, Choueiri's research involves developing neurotechnologies to map the molecular interactions of the brain, to reveal the fundamental mechanisms of brain function and repair dysfunction.
This is argued to represent the dysfunction and slow repair of an online verbal associative process in the brain following inadequate sleep.
Finally, the 5-HT1A recepresentresent in the cingulate cortex, as demonstrated in the present study, may constitute an important target for drugs used to repair dysfunction of glutamate neurotransmission, which is observed for example in schizophrenia.
Telomere dysfunction leads to repair by non-homologous end joining or homologous recombination that can induce chromosome or chromatid fusions [8], [53].
