Sentence examples for dysfunction proof from inspiring English sources

Within minutes of its passage, congressional leaders of both parties and the president rushed to the television cameras to praise each other for a bipartisan bill that they claimed signaled the end of dysfunction; proof that Washington can work.

Restless legs syndrome, non-specific urinary incontinence and erectile dysfunction exposed in the "proof of no benefit" section were side effects of ADs.

The absence of a clear proof of biological dysfunction underlying neurological symptoms should not lead automatically to the diagnosis of a conversion disorder.

There is so much proof about endothelial dysfunction that it is reasonable to believe that there is diagnostic and prognostic value in its evaluation for the late outcome.

The response to rGH in KSS is somewhat unpredictable, similarly to cardiac dysfunction, and there is no proof that rGH treatment contributes to the onset of the conduction block.

Although direct proof of mitochondrial dysfunction and its correlation with the strong ion gap is not yet available, Bruegger and colleagues' findings support the concept of early mitochondrial dysfunction and energy debt during hemorrhagic shock: in fact, in critically ill patients, the strong ion gap was a strong predictor of mortality if it was the major source of metabolic acidosis [ 34].

Dynamic P MRS showed PME (fructose-1-phosphate) increases and decreases in the levels of ATP and Pi in healthy volunteers in response to bolus fructose administration, whereas in three adults with disorders of fructose metabolism no spectral changes were seen, providing proof of fructokinase dysfunction [ 107].

In view of the therapeutic promise of upregulation of CSPα and the undesired consequences of CSPα dysfunction, our data establish an essential proof of principle that pharmaceutical agents can selectively target the neuroprotective J protein CSPα.

So, there seems to be a consistent proof of principle that endothelial dysfunction can be normalized by intervening postprandial hyperglycemia.

Nonetheless, our data represent a proof-of-concept of diabetic BM dysfunction in humans.

However, formal proof of this indispensable role of DC dysfunction in SpA pathogenesis still needs to be established in non-transgenic animal models or using adoptive transfer experiments.