Sentence examples for dysfunction growth from inspiring English sources

Furthermore, elevated levels of the anti-angiogenic factor sFlt-1 (sVEGFR-1) have been causally implicated in placental dysfunction, growth restriction and fetal loss [32], [37].

Over production of ROS causes cellular damage, dysfunction, growth inhibition, and so on.

Molecular indicators of glutamate excitotoxicity, impaired axonal transport, oxidative stress, mitochondrial dysfunction, growth factor deficiency, protein aggregation, abnormal RNA metabolism, and apoptosis have been observed in human brain, spinal cord, and transgenic disease models long before symptom onset.

These results demonstrate that ICV injection of ASO-20-37 ASO-20-37 ASO-20-37otor dysfunction, growth impairmelicitedshortened lifespan in a dose-dependent motorr, even in adysfunctionsimilar to what we previously observed in neonatal mice (Sahashi et al, 2012).

In hydrocephalus, the most important specific functional concerns are: · Impaired mobility and ambulation (e.g., cerebral palsy) · Impaired cognition (mental delay, behavior) [ 31- 33] · Sensory deficits (vision, hearing) [ 34- 36] · Endocrine dysfunction (growth, puberty, weight balance, fertility) [ 4, 37, 38] · Epilepsy [ 8, 39] · Depression [ 37] · Pain (chronic headache) [ 40].

Promising research has been conducted in recent years that have the potential to affect the development of new treatment approaches for salivary gland dysfunction including growth factors, gene transfer, artificial salivary glands, and stem cell transplantation (reviewed in [ 17]).

Inadequate maternal and/or fetal micronutrient supply could compromise any of these processes, impairing angiogenesis and leading to placental dysfunction, fetal growth restriction (FGR), and preterm birth (PTB) (10).

EOPE is considered a more severe, mostly fetal disorder, and it is typically associated with inadequate placental implantation and subsequent placental dysfunction, intrauterine growth restriction, low birth weight, and adverse maternal and fetal outcomes, including perinatal death.

González et al. have studied vaspin gene expression regulation in WAT of mice under different physiological (diet, pregnancy, age, and gender) and pathological (gonadectomy, thyroid dysfunction, and growth hormone deficiency) conditions, associated with energy homeostasis and insulin sensitivity disorders.

Key abnormalities that were observed in these different studies included reduced fertility (including gonadal dysfunction), impaired development and slow growth.

Biomarkers included measures of inflammation (interleukin-6; IL-6), cardiac cell damage (high sensitivity troponin T; hsTnT), endothelial dysfunction (vascular endothelial growth factor; VEGF), and overall cardiac prognosis (N-terminal pro-brain natriuretic peptide; NT-proBNP).