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Objective: Transient left-ventricular dysfunction because of myocardial reperfusion injury is a significant problem after cardiac surgery, but the underlying complex pathophysiology is still poorly understood.
However, alterations in the pelvic floor muscle intrinsic structure that lead to muscle dysfunction because of childbirth and aging remain elusive.
Treatment with pelvic external beam radiotherapy with brachytherapy (EBRT/BT) for gynecological cancers may cause sexual dysfunction because of vaginal shortening and tightening.
These procedures had the potentials to induce interstitial lung edema and subsequent pulmonary dysfunction, because of encouraging inflammation of lung tissue, hemorrhage, prolonged surgery, and increased transfusion.
Some of the participants explained their sexual dysfunction because of vaginal dryness.
Unilateral urinary obstruction does not normally cause acute renal dysfunction because of compensation by the contralateral kidney.
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First, structural changes are common indicators of embryonic dysfunction, because 60% of lethals exhibit abnormal anatomy, which can be detected by simple observation.
There is a strong relation between CECs and endothelial dysfunction because numbers of CECs correlate with surrogate markers of disturbed endothelial function such as flow-mediated dilation and von Willebrand factor (vWF) [ 12].
Moreover, it has been reported that DM-related complications or patient characteristics such as diabetic nephropathy, diabetic retinopathy, obesity, insulin resistance, or autonomic neuropathy are associated with subclinical LV myocardial dysfunction, because all of them are considered to be accumulative phenotypes of poor metabolic control [ 11- 17].
Second, although this study included patients older than 70 years, we did not evaluate postoperative cognitive dysfunction because none of these patients required prolonged ICU stay due to postoperative cognitive dysfunction.
We can hypothesize that the accelerated decrease of VEGF and PEDF genes expression at the mRNA level in OXYS rats is due to early RPE dysfunction because both of these genes are primarily expressed in RPE cells.
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