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Independent from exposure to DEHPlow or DEHPhigh house dust, we found no significant changes in the concentrations of cytokines and ECP in nasal secretions in healthy subjects.
We also compared linear slope estimates with Meeker (estimated from Figure 2A), finding a larger slope (8.77 × 10 5 ng/mL per ng/g in this study versus 6.66 × 10 6 ng/mL per ng/g in Meeker et al).. Therefore, for every 10 ng/g increase in TDCIPP concentration in house dust, we found a 0.877 pg/mL increase in its metabolite in urine.
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Taking inspiration from the orbital dynamics of dust, we find that spacecraft length scaling is a means of enabling infinite-impulse orbits that require no feedback control.
Looking at the relationship between estimated gas-phase air concentrations and dust concentrations, we found a significant positive correlation across all compounds (τ =0.37, p < 0.05).
We found dust-covered offices with decades-old bureaucratic paperwork scattered across the floor, a clocking-in machine for the workers, and a plethora of drill belts and other industrial tools.
By studying the light curves of the halo of Cyg X-1, we found a dust concentration at a distance of 1.75 kpc from the Earth.
Chlorpyrifos was measurable in the house dust of all homes, whereas we found parathion in only 41% of the homes.
However, consistent with other studies [ 15, 36], we found that dust serves as a stable matrix and indicator of potential indoor exposure for some pesticides.
When restricting to those exposed to wood dust at a substantial level, we found an increased risk in two of three subsets of the data.
We found that dust wipe samples taken from homes with chipping, peeling, or flaking (i.e., deteriorated) paint had higher windowsill PbD than homes without deteriorated paint, which is consistent with other research (U.S. HUD 2004; Wilson et al. 2006).
We found that carbon dust inhalation caused bronchoconstriction in healthy humans and in anesthetized cats [ 31].
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