This suggests that increased ACh levels during the retention session are strictly due to aversive memory formation.
The spectral theta power extremely decreased just before sFSTG during the retention interval.
The flow field around the aggregate during the retention is evaluated using the µPIV technique.
In addition, there were some animals that received a shock during the training phase that did not make contact with the probe during the retention phase.
Furthermore, unlike Experiment 1, the probe was electrified for both groups during the training session and was not electrified during the retention session.
(b) Fear-conditioning memory, expressed as % freezing during the retention test, was significantly impaired in both vehicle- and IL-1ra-treated Tg2576 groups, ie, the IL-1ra treatment had no effect on memory impairment in this task (n=9/group).
Statistical evaluation of the different groups during the retention session with an overall two-way ANOVA revealed significant interaction (F48,228=11.30, p<0.001) and treatment (F4,228=7.39, p<0.05) effects.
Experiment 2 evaluated whether a process of fear incubation, developed during the retention interval but obscured by a ceiling effect, produced the attenuation of LI.
Hippocampal ACh levels were only significantly increased during the subsequent retention session, if the animals made contact with an electrified probe during the training session regardless of whether the probe was electrified or not during the retention session (Figure 2).
Most animal models currently used to study the retention of implants in bone are influenced by shear forces introduced during the retention test.
Although neurochemical differences between the two groups were not detected, they did differ behaviorally, in that the shock off shock on group, but not the control group, displayed burying behavior during the retention session (data not shown).
