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In addition, the experiment was performed using different kind of polyclonal antibodies, targeted against HPBP (Figure 2), and targeted against the very conserved N-terminal peptide of DING proteins (supporting document – Figure S1).
For example, it is known that over-expression of c-myc protein leads to increased cellular proliferation via amplified rates of ribosome biogenesis [15] and that myc is over-expressed in 70% of colon cancers [16], consistent with the P-rich signature that we document (Figure 1).
Reduced Cross-Lagged Monozygotic Twin Difference Model for Author Recognition Test (Reading Exposure), Reading Ability, and General Intelligence Differences (for Saturated Model, see Figure 3 in the Main Document) Figure S5.
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We conclude the result section with a network representation of the topical landscape of the analysed documents (Figure 7).
This fact entails the reduction of the number of received documents (Figure 3a) and the increment of the delay (Figure 3c) and the percentage of timeouts (Figure 3d).
For each excluded study, an appropriate reason was documented (Figure 1).
The TBSS results shown in coronal slices are provided in the supporting documents (Figure S2).
Fifty-one (26%) patients had macular lesions, 42 (21%) had nodular lesions, 10 (5%) had fungating lesions, and 98 (50%) did not have lesion morphotype documented (Figure 2).
Thus, the percentage of ALDH+ cells was not changed in the treated tumours compared to untreated controls (Figures 6B and S5B), even though inhibitory effect of DTIC on tumour growth/volume was documented (Figure 5C).
All had discrepancies between the two documents (figure, box 2).
The postoperative gonioscopic appearance of the cleft was also documented (figure 4).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com