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These regional changes in chromosome copy number were particularly obvious when heat maps generated from 468GFP and 468LN DNAs were compared to a normal reference female Yoruba (YRI) population (Ibadan, Nigeria; [25]) from the International HapMap Project (Figure 3).
Tumour DNAs were compared with DNAs from 167 anonymous healthy controls, which were provided by the IZKF Z3 Core Unit Genomics.
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Reported symptoms and objective clinical findings of women with and without HPV DNA were compared by multivariate analysis.
Results obtained with the precipitated DNA were compared to those from input DNA samples.
In Fig. 5, nanoparticles formed using JetPEI™ and plasmid DNA were compared with injections of plasmid DNA alone.
HIV-1 pol nucleotide sequences covering the PR/RT region derived from plasma viral RNA and cellular DNA were compared to determine any correlation between the two.
Viral divergence values, number of dN/dS sites and entropy between sequence sources (plasma, PBMC, cervical RNA, and cervical DNA) were compared using Wilcoxon Rank Sums tests (JMP, Cary, NC).
Known positions conferring resistance to antiretroviral therapy from HIV-1 pol nucleotide sequences covering the PR/RT region derived from plasma viral RNA and cellular DNA were compared for each sample, to determine any correlation between the two.
The banding patterns of DNA were compared visually.
Germline DNA, obtained from a blood sample, and tumour and xenograft DNA were compared.
Noncovalent interactions of brostallicin and tallimustine (TAM) with DNA were compared to those of distamycin A (DISTA).
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