Sentence examples for dna can trigger from inspiring English sources

But at this point, it's not clear whether it's one gene or several that raise the risk of disease, or how deletions and duplications of the same piece of DNA can trigger outcomes as different as schizophrenia and mental retardation.

With the presence of three ingeniously designed hairpin structures and Exonuclease III (Exo III), the target DNA can trigger two independent cycles of reactions: hairpin assembly reaction and Exo III cleavage reaction, which are designed to initiate target DNA recycling amplification and reporter DNA amplification, respectively.

In addition, low quality FFPE DNA can trigger allele dropouts and produce inaccurate results [ 6, 15].

The different associations of TFs with their cis-regulatory elements on the DNA can trigger, counteract or modulate these regulatory states of genes.

Brain cells that inherit damaged DNA might not function correctly, while serious breaks that affect both strands of the DNA can trigger a response that kills the mutated cell.

Silencing of tumor suppressor genes by DNA methylation provides a powerful molecular mechanism by which DNA methylation can trigger cancer, and also provides a rationale for therapeutics aimed at inhibition of DNA methylation and re-expression of silenced tumor suppressor genes.

It has been shown that stem cell niches in Arabidopsis are hypersensitive to DNA damage, and an even a low dose of DNA damage can trigger programmed cell death selectively in these stem cells (Fulcher and Sablowski 2009).

Besides these direct effects of oxidative injury, there can be indirect injury because the nicks and breaks in DNA strands can trigger activation of Poly(ADP) polymerase (PARP), which alters gene expression, DNA replication and may trigger apoptosis.

While in the presence of target DNA, it can trigger the self-assembly process of P1 and P2 and initiate the Exo III-assisted two recycling process, resulting in the formation of G-quadruplex structure on electrode surface.

Chrysotile-induced DNA damage can trigger apoptosis in various cell types following 3 to 4 h of exposure [8], [10].

This is important because a stalled DNA polymerase can trigger the intervention of LBPs during S phase repair with or without a non-scheduled DSB and with or without the involvement of recombinational repair (Fig. 3 in Symington [56]).