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The divergence is probably due to the fact that the plasmids encoding ubiquitin mutants used in the two studies are different.
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This difference is probably due to the overexpression of Baz.
This divergence from the other infection models is probably due to AidA, a protein regulated by quorum sensing and one of the major virulence factors for C. elegans pathogenicity [43].
The presence of SNPs in glutamate dehydrogenase is probably due to a gene duplication followed by divergence; our preliminary assembly of ~4× coverage of the S. salmonicida genome indicates distinct upstream sequences for two paralogs (unpublished data).
This is probably due to the difference in receptor distribution.
This is probably due to chemical reactions in the flame.
This effect is probably due to enhanced GABA activity.
It is probably due to the lack of added sugars.
This is probably due to two reasons.
This is probably due to adaptor redundancy.
This is probably due to overfitting.
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