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It might, however, disrupt identification of genes that are removed from carriage isolates for adaptation to the invasive environment, if such genes exist.
Rearranging parts of an object can disrupt identification of the object, while leaving low-level features still present.
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If different backgrounds are present during testing, this does not disrupt the identification of particular objects previously trained, because the different backgrounds are not associated with the object to be recognized.
Unlike genomic studies, transcriptomic data vary according to environmental conditions, and a small number of highly expressed genes can disrupt the identification of other more infrequently expressed genes [ 14].
High-minded plans for erecting a Museum of Liberty on the grounds of the nation's original Executive Mansion are disrupted by the identification of the quarters occupied by George Washington's slaves.
We repeated the homogeneity test for partitions as used in tree reconstruction, if base pairs were disrupted by the identification of the corresponding partner as randomly similar (ALISCORE), remaining formerly paired positions were treated as unpaired.
Using experimental animal models where this transcription factor was disrupted allowed the identification of dependent genes that are critical in pulmonary protection and confirmed that its disruption promotes susceptibility to several prooxidant-induced lung diseases, primarily owing to decreased levels of the basal and inducible expression of several antioxidant enzymes [ 37– 40].
As the head of drug discovery, she supervised projects involved with the identification of compounds that disrupt RNA-protein interactions with a view to developing antibiotics.
SNP_search broadens the definition of an exon to include 10 nucleotides upstream and downstream of an exon, to facilitate identification of mutations that disrupt splice sites.
Initial methods focused on the identification of mutations that disrupt transcription factor binding sites (TFBS) [ 13- 15].
On the basis of mapping data, the identification of lesions that disrupt the splicing or expression level of notch3 and complementation analysis, we conclude that both the st51 and zm mutations disrupt the function of notch3.
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