Among Clinical students only men displayed a small increase (time coefficient 0.33 (95% CI 0.11 to 0.55)).
However, addition of clinical variables to the model displayed a small increase in the power of the model.
Indeed, even though UbI44A-EEA1 was expressed at a higher level than Ub-EEA1, as demonstrated by immunoblotting, cells expressing this mutant only displayed a small increase in size of endosomes, comparable to cells expressing EEA1.
FP 0.5 (not shown) and 1 mg·mL−1 treatment did not significantly alter this response with significant bronchoconstrictions still present at 10 min. When challenged with LPS alone, vehicle-treated groups displayed a small increase in bronchoconstrictor response to histamine significant at 10 min compared with pre-challenge.
As shown in previous studies (Kwiatkowska and Dumais, 2003; Kwiatkowska, 2004; Kwiatkowska and Routier-Kierzkowska, 2009), the quantification of growth anisotropy shows that cells in the boundary displayed a small increase in length only in the direction parallel to the border between meristem and primordium, suggesting compression by the growing organ (Hamant et al., 2008).
ILK-deficient platelets, however, displayed only a small increase in fibrinogen binding.
TRAIL-resistant cells exhibited a strong TRAIL-inducible NF- κB activity, whereas TRAIL-sensitive cells displayed only a small increase in NF- κB-binding activity.
Animals with substantial SE showed huge overexpression of TSPO in vitro in relevant brain regions such as the hippocampus and amygdala (P < 0.001), while animals with mild symptoms displayed a smaller increase in TSPO in amygdala only (P < 0.001).
In cells that displayed this rapid voltage response, a small increase in stimulus strength usually evoked a full-blown AP.
