Sentence examples for disorder profile from inspiring English sources

Intrinsic disorder profiles of existing cIFNs-α possess remarkable similarity to the consensus disorder profile calculated as averaged disorder predispositions of all human IFNs-α.

Correct naming of schizophrenia, depression and eating disorder was significantly more common in the intervention group at pre-test, while the anxiety disorder profile was equally unrecognized in intervention and control group.

First of all, raters get the point that some people, whilst clearly disturbed and stressed as a result of life-long patterns of maladaptive behaviour, such as Aileen Wuornos and Sarah Morton, do not fit any single prototypical personality disorder profile [ 26- 28].

Fig. 3C E show disorder profiles generated for these arrestins by several common disorder predictors.

Furthermore, a strong correlation is established between the peculiarities of disorder profiles of these proteins and their known structural features.

Often, in corresponding disorder profiles, MoRFs are indicated as "dips"; i.e., regions with locally increased order propensity within long IDPRs.

If the peculiarities of disorder distribution within the protein sequence are related to protein functionality, then comparison of the disorder profiles of artificial cIFNs (query profiles) with the averaged disorder predisposition profile of human IFNs-α (target profile) can be used in the design of novel cIFNs.

In each plot, red line shows the mean disorder propensity calculated by averaging disorder profiles generated by six common disorder predictors: PONDR® VLXT (Dunker et al., 2001), PONDR® VSL2 (Peng et al., 2005), PONDR® VL3 (Peng et al., 2006), PONDR® FIT (Xue et al., 2010), and IUPred_short and IUPred_long (Dosztanyi et al., 2005).

An intrinsic disorder score for each position in the sequence was computed based on intrinsic disorder profiles generated with DisEMBL [30], IUPRED [31], RONN [32] and FoldIndex [33].

The intrinsic disorder profiles (FIG. 8) show a high flexibility of the S240-P254 linker between the KIM region and the catalytic domain of PTP-SL, supporting thus this hypothesis.

A comparison of the disorder profiles of these proteins and the corresponding proteins from angiosperms belonging to the same subfamily shows that the N-terminal region is intrinsically disordered.