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Numerous inflammatory diseases display elevated interleukin (IL -8, and most are assocIL -8 with andeactive stromost
Most diseases display a 'pathological footprint' on a systems biology level, even if the observed phenotype is localized to a tissue or organ.
In addition, human renal biopsy samples from patients with a variety of fibrotic kidney diseases display cells within the tubular structures that exhibit both epithelial and mesenchymal features [64, 65].
Many of these diseases display gender preferences in presentation or altered severity that occurs with puberty, the mechanism by which is unknown.
These Gram-negative bacteria, responsible for a large portion of antibiotic-resistant bacterial diseases, display a complex cell envelope comprising an outer membrane and an inner membrane delimiting the periplasm [6].
All of these pulmonary diseases display distinct gender preferences in presentation or a change in disease severity that occurs at puberty, the cause of which is unknown [14] [17].
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For example, rasagiline, approved in 2006 for neurodegenerative diseases, displayed a multi-target profile.
Willis et al. [3] suggested the use of a common term: white syndrome (WS), for Indo-Pacific scleractinian coral diseases displaying acute tissue loss exposing white skeleton in the absence of other disease signs or established causation.
Only respiratory diseases displayed the opposite trend.
Current studies on understanding human disease states led researchers to think that there was increasing evidence that heterozygous null alleles led to more and more human diseases, displaying the importance of haploinsufficiency in disease.
As with many other infectious diseases displaying seasonal patterns (11), the mechanisms driving the predictable seasonal periodicity of meningococcal disease are not well understood, and the specific effects of environmental factors on case occurrence remain unclear.
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