Exact(1)
Except for one study in which children with cyanotic-corrected heart disease (transposition of the great arteries, tetralogy of Fallot) had low maladjustment rates [ 20], all rates were significantly higher than those reported for either a normative group or another type of control group (i.e., siblings or healthy controls).
Similar(59)
The most common congenital conditions at risk for progressive right ventricular dilatation and dysfunction are pulmonary valve stenosis (PS), repaired tetralogy of Fallot (ToF), chronic pulmonary regurgitation, intra- or extra-cardiac shunts, Ebstein's disease and transposition of great arteries with systemic RV (Figs. 2, 3 and 4) [11, 12] 12].
A 46-year-old man was born with complex congenital heart disease including transposition of the great arteries, situs inversus, atrial and ventricular septal defects and stenosis of the pulmonary outflow tract.
Their further study should enrich our understanding of infection and disease processes, the diversity of transposition mechanisms and regulation, and of genome evolution.
In patients with congenital heart disease, 5 of those had transposition of great arteries (2 were congenitally corrected), and 1 had coarctation of the aorta with dysplastic aortic valve and previous surgeries with resulting severe left ventricular (LV) dysfunction.
In the 28 cases of undiagnosed duct dependent heart disease in the other referring regions, transposition of the great arteries constituted 39%, simple coarctation of the aorta 26%, and complex coarctation 18% (table 5).
Other rarer causes are related to postoperative complications of congenital heart disease such as following repair of D-transposition of the great arteries (TGA).
To assess the effect of prenatal diagnosis of congenital heart disease on neurocognitive outcomes in children with d-transposition of the great arteries (TGA) after surgical correction.
The current surgical treatments for this disease aimed at hip preservation have attempted to induce revascularization or transposition of the necrotic area [ 9– 11].
However, active transposition by LINEs does occur in humans and occasionally causes disease when the insertion occurs in an exon [ 8], splice site, or regulatory region [ 11].
The underlying disease was myocarditis in two patients, dilatative cardiomyopathy in one patient, d-transposition of the great arteries in one patient, and undetected Bland-White-Garland syndrome in one patient.
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