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The proportion of mutant mitochondria determines both disease expression (whether it will affect offspring) and disease severity.
The disease expression and the mechanisms involved are poorly known.
Pig genetic background has been pointed to influence disease expression.
Differences of disease expression among trials were associated with the environmental factors prevailing during the experiments.
Disease expression has been highly variable in both incidence and severity, and symptoms have not recurred every year.
The difference in disease expression between naturally infected vegetation in New Zealand and the northern hemisphere is striking.
Crucial topics include fragmentation effects on pathogen spread and geophysical features that predispose forest patches to disease expression.
The disease is endemic, causing a chronic yield loss. Disease expression is complex as incidence and severity are affected by drought, soil and nutrient conditions.
This incomplete penetrance of PAH suggests multi-faceted genetic, epigenetic and/or environmental factors may affect disease expression (Ma and Chung, 2014; Austin and Loyd, 2014).
The phenotypic disease expression consists in degeneration and infiltration of the muscular tissue characterized by replacement of myofibers with adipose tissue.
Therefore we focused on delineating the relationship between psychosocial functioning, cognition and brain structure, in relation to disease expression and genetic risk for BD.
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