Not only are housing prices in the biggest cities moving in a different direction to those in smaller centers, there is also a glaring discrepancy in the amount of development being undertaken.
To support this model, we wished to exclude the possibility that the decreased mRNA levels of the plastid genes are a simple reflection of discrepancies in the apicoplast development, particularly apicoplast DNA replication that is rapidly ongoing during the schizont stage.
Overall the discrepancy in microstructural development beyond 90 days in the two concretes would explain the contradictory performance over the longer time frame.
Furthermore, the overall discrepancy in colony development (more and bigger terminal branches) may be interpreted as a response to the water drag forces acting upon the colony, yet colonies in shallow areas are exposed to higher water flow and vice versa.
A possible explanation of this discrepancy could be the development of secularism in the last 25 years in Switzerland, as well as in the majority of European countries [ 28].
The reason for this discrepancy may lie, in part, in the development of adjuvant chemotherapy.
In our experiments, the discrepancies in infection status and in the development of clinical signs in infected but clinically healthy birds could be a result of several host factors (e.g., age, immune status), virulence, and adaption of the inoculated ABV suspension to the cell culture.
The reason for this discrepancy in the timing of mammary tumor development is unclear and is in our opinion not only due to the mixed (FVB/N/SV129) genetic background as was suggested because we also observed a long latency in a full FVB/N background.
The discrepancy in development of stria vascularis between Slc26a4+/− and Slc26a4−/− mice was most apparent at P3 (Fig. 11).
