Sentence examples for diffusion of the complexes from inspiring English sources

Bulk diffusion of the complexes, charge-transfer reactions and the inhibiting nature of the film formed were thoroughly investigated.

An excess of free CD, especially the bulky HP-β-CD, made the diffusion of the complexes in the relatively low mesh size 2% polymer network more difficult.

ED for both complexes in ILs are found slightly larger compared to the activation energies of viscous flow (Eη) for the corresponding IL, indicating that the viscosity is not the only factor determining the diffusion of the complexes.

The activation energies for the diffusion coefficients were close to that for the viscosity of the ionic liquid, indicating the viscosity is a major factor that determine the diffusion of the complexes.

In the considered operating domain, the diffusion of the complex reaction product, protonated-amine-carbamate, could be considered non-limiting.

The rate of monolayer formation appears to be controlled not by mass transport or interfacial binding but by surface diffusion of the complex.

Whereas at 24°C the reaction is sufficiently rapid that the current exhibits a significant kinetic component, at 2°C the equilibrium is effectively frozen and the limiting current is controlled entirely by diffusion of the complex from bulk solution.

Alternatively, a diffusion-based mechanism has been proposed, where a gradient of ParA molecules bound to the chromosome interacts with a parS/ParB complex, directing the diffusion of the complex.

To interpret what this means in terms of the oligomeric state of SP-A within various micelles, we have estimated the contribution of the micelle itself to the diffusion of the complex and used this to estimate the oligomeric state of the SP-A within the micelle SP-A comicelle SP-A

As the APC/C is not specifically localized within cells, although it is enriched on the spindle, at spindle poles (Huang and Raff, 2002) and centromeres (Acquaviva et al, 2004), it is widely held that the diffusion of this complex from the kinetochore into the cytoplasm is critical for forming the inhibitory MCC APC/C complex (Sudakin et al, 2001; Herzog et al, 2009).

This is because this mechanism is only governed by the diffusion of the partition complex.