In this work, scaling relations are constructed for diffusion induced damage in intercalation electrodes based on an extensive set of numerical experiments with a particle-level description of microcrack formation under disparate operating and cycling conditions, such as temperature, particle size, C-rate, and drive cycle.
To answer these questions, a finite element model was developed that describes deformation, diffusion and damage in a cross-section of skeletal muscle tissue.
Exchange of costly intermediates is associated with a significant risk, as exported metabolites can be lost through diffusion, chemical damage, or cheating (Dobay et al., 2014; Oliveira et al., 2014; Wintermute and Silver, 2010).
In fact, although control cells evidenced a vesicular pattern of LY fluorescence (index of healthy lysosomes), hPrP90-231-treated cells showed diffuse cytosolic fluorescence, indicating LY diffusion through damaged lysosomes.
Applying metallography to cross-section microscopic slides, diffusion subsurface damages were studied caused by the cutting.
Thus, the fast GTPase activity (or high affinity to GTP) of PsbO2 might facilitate rapid disorganisation of PSII-LHCII supercomplexes and crystalline arrays allowing rapid diffusion of damaged PSII out of the grana and their redeployment after D1 replacement.
The diffusion lengths and damage coefficients are also extracted by modeling the quantum efficiency.
The results suggest that further damage diffusion could in principle be achieved by additionally modifying the interface between tiles.
The polymer's penetrant uptake can be described well by regular diffusion, with nuclear damage centres acting as saturable traps.
The results show that the presence of discontinuities and hierarchies promotes stable energy dissipation before failure, ensures damage diffusion throughout the specimen, and delays damage localisation in otherwise brittle composites.
Theoretical Monte Carlo simulations suggest that changes in tissue water diffusion following tissue damage are predominately attributable to alterations in the volume and tortuosity of the extracellular space.
