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How can the deficit in the executive function network be related to the arithmetic difficulties in developmental dyscalculia?
Moreover, several researchers have suggested that some of the difficulties in developmental dyscalculia may be related to attention [e.g., see [ 20]].
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Her phenotypes included feeding difficulties in infancy, developmental delay, seizures, microcephaly, strabismus, hypertelorism, a low hairline, ear malformations, a broad nasal bridge, wide mouth, thick lips, and prominent incisors.
The second type of evidence supporting our claim that difficulties with FCs in developmental biology are the primary obstacles for students learning evo-devo is the relative prevalence of different types of conceptual difficulty.
The aim of the present study was to analyze handwriting difficulties in children with developmental coordination disorder (DCD) and investigate the hypothesis that a deficit in procedural learning could help to explain them.
Some of the studies that were described above did not differentiate between pure developmental dyscalculia and the co-morbidity between developmental dyscalculia and ADHD [e.g., [ 13, 28, 36, 38, 40]], thus it is not clear whether the attentional difficulties in those with developmental dyscalculia are part and parcel of their dyscalculia or of the co-morbid deficit (i.e., ADHD).
19 Children with moderate developmental delay, despite the same initial difficulties, have great variability in developmental trajectories, with some children catching up while others exhibit evolving difficulties with age.
This is probably because of the technical difficulties in tracing the continuous developmental changes at different stages of mammalian embryos.
However, our findings differ from nationally representative samples [ 14] of children with developmental difficulties in which Medicaid enrollment did not influence receipt of services.
Additionally, research has suggested a close interrelation among developmental difficulties in the child and progress of parental chronic illness within the family life cycle [ 32].
The rapid elongation of the inflorescence stem in the model plant Arabidopsis thaliana is accompanied by radical changes in cell wall structure and chemistry, but analysis of the underlying mechanisms and identification of the genes that are involved has been hampered by difficulties in accurately sampling discrete developmental states along the developing stem.
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