In general, differentiation occurs in a proximal to distal sequence, with the tracheal epithelial cells differentiating first.
The thymocytes then move to the medulla of the thymus, where further differentiation occurs.
Terminal differentiation occurs not only in embryonic development but also in many tissues in postnatal life.
The differentiation occurs in an upward direction, so that the maturation of the vascular tissues follows at a more or less constant distance behind the apex.
The question of how much genetic differentiation occurs during speciation has become answerable only with the relatively recent development of appropriate methods for comparing genes of different species.
Tracheary element differentiation occurs synchronously at high frequency above initial cell densities of 4.2 × 104 cells mL L−1, but is significantly suppressed below this threshold.
In sepsis, impaired function and loss of antigen-presenting cells are observed in secondary lymphoid organs [1], the site where antigen-dependent B cell differentiation occurs in health.
Overall, the data demonstrate that modulation of MSC differentiation occurs at the transcriptional level and gene expression of bone-related proteins is differentially regulated by the ECM.
Differentiation occurs within groups, represented as branches in the cladogram.
How Tfh differentiation occurs is currently unresolved.
These cells migrate to the spleen where differentiation occurs.
