Sentence examples for differential sensitivity for from inspiring English sources
However bacterial strains showed differential sensitivity for each extract.
We extensively addressed the question of whether FOBT, the most common non-invasive tool for CRC screening, shows differential sensitivity for detecting left- vs right-sided advanced colorectal neoplasia within a large screening study conducted in average-risk subjects.
Briefly, PLIER exhibits higher signal reproducibility and differential sensitivity for low expressing genes yet the variance can be unstable on a log scale, whereas RMA demonstrates fold change compression at the low end of expression, but the variance is stable on a log scale [ 19].
The apparent differences in anti-phospho-antibody reactivity to the in vitro phosphorylated proteins shown in Figure 1C do not represent a differential sensitivity of the antibodies for the different alanine mutants but more likely the variable stability of the alanine mutant proteins.
At this juncture, it seems unlikely that differences in the ability of vorinostat to inhibit HDAC activity or regulate the expression of key genes are the primary hypothetical mechanisms for differential sensitivity of tumour and normal cells to this agent.
This study was designed to determine the molecular basis for the differential sensitivity of the vasculature to NO.
Further studies are also needed to identify the molecular basis for the differential sensitivity of medial and lateral mDN populations to Shh chemoattraction.
To investigate the basis for the differential sensitivity of cancer cells to inhibitors of the V-ATPase, we have made use of the observation that cells often respond to stress by up-regulating critical components of pathways that are sensed to be failing, as for example the response to the failure of protein folding in the endoplasmic reticulum [19].
Little is known so far about the differential sensitivity of the cell lines, for example, with regard to oxidative stress.
Such evidence provides support for the differential sensitivity of these factors to associated physical and psychological states.
Comparison of the FGFR4KV550L FIIN-2 and FGFR4KWT ponatinib structures explains the molecular basis for the differential sensitivity of these two inhibitors toward the V550L gate-keeper mutation.
