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This bias was not likely to be differential by age group and thus should not affect the overall picture of the results.
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Looking at differentials by age group, we noted that older women (30 49 years) were more likely to be anemic than younger women (15 29 years), 43.6 % and 38.6 %, respectively; the association was statistically significant (p < 0.001).
In neither case is there a clear reason to expect these misreports of own age to be differential by relationship age disparity, and thus the presence of such errors should again make our findings a 'worst case'.
Differential effects by age were documented by Hoynes et al. (2012), whereas Clark and Summers (1981) found no such evidence in the age span of 20 64.
The pattern of differential susceptibility by age is presented below.
Our assumption of a differential susceptibility by age among women was necessary to fit the observed patterns.
We built on this observation to test the impact of differential susceptibility by age with our model.
Hence, these main drivers of prevalence have a differential impact by age.
There was no differential effect by age, sex, or BMI group.
Filtering for differential expression by age (ANOVA, FDR 5%, and fold change >1.5) resulted in 7,274 unique genes.
Pre-planned subgroup analyses did not show any significant differential effects by age or history of the previous fall.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com