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In contrast, LS in advanced LS+ UGCs in cluster B (A107, A108 and A110) was virtually indistinguishable morphologically but showed genomic constitutions different from LS in cluster A. This may be a kind of phenocopy; a fraction of LS+ UGCs were considerably similar in genomic profile to LS−/TC+ UGCs.
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For example, corticosterone response to restraint and pain perception were identical in absolute values but different in variation; conversely, although H-CORT mice showed a differential corticosterone response compared to both the other groups, their variation was identical to AFR but different from L-CORT.
This may well reflect that L-FUΔ24 plays a biological role different from L-FU.
However, sensitive cell proliferation treatment with ZJW and L-OHP were not found to be significantly different from L-OHP control group.
For the QTL centered around position 78-80 on chromosome 5, some the DOG17 NILs (Alonso-Blanco et al. 2003) were found to be significantly different from L er for HGI and several of the other measured traits (Table S3).
UCr excretion rate 10 hours post-transplant had numerically the largest area under ROC curve for discrimination between IGF and SGF/DGF groups (although not significantly different from L-FABP and NGAL).
For (K,Linmathcal{S}_{o}^{n}), (pgeq1), and (lambda,mugeq0) (not both zero), the p-harmonic radial combination (lambdaast K+_{-p}muast Linmathcal{S}_{o}^{n}) is defined by (see [17]) rho(lambdaast K+_{-p}muast L,cdot)^{-p}=lambdarho K, cdot )^{-p}=lambdarho Kt)^{-p}. Note that here '(varepsilonast L)' is different from '(varepsiloncdot L)' in Firey p-linear combination.
This functional effect on inversion and eversion was differentially specified along the width and length of the foot such that F-L was always different from M-L and F-M.
Stages L-II and L-III as well as stages L-IV and L-V are similar to one another, but very different from stage L-I.
In our observations of the effect of temperature, pH, ionic concentration, and the denaturation agents, only the thermal behavior of d-EAK16 is different from its L-peptide counterpart.
We noted that increased β-dystroglycan did not protect TA muscles after treatment with verapamil and diltiazem, suggesting either that the mechanism of action for streptomycin is different from the L-type calcium channels blockers or that restoration of β-dystroglycan expression alone is not sufficient to stabilize the membrane.
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