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The differences in low temperature toughness with respect to the processing history appear to stem from differences in the reactive nature of these two types of compatibilizers.
Notwithstanding the somewhat greater reactive-centre diversity of the Arabidopsis serpins compared to that of rice, the substantial differences in the reactive centres of the rice serpins are reminiscent of the oat-grain serpins [ 32] and unlike the glutamine-rich reactive centres in serpins of wheat [ 34] and rye [ 54] grain.
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The differences in the reactive-centre sequences of these otherwise closely related serpins might reflect a need to broaden the inhibitory specificity of the rice serpin complement for defence against digestive proteinases from insects or pathogens.
The difference in the reactive potential of Sn and Si with lithium was favorable for reducing the mechanical stress of the active hosts.
However, this difference in the reactivity was detectable only when reactive enzymatic intermediates were involved in the biodegradation process and were negligible for the biodegradation process realized via HOCl-driven reactions.
Further research showed that the enantioselective effects of CILs resulted from the differences in the production of reactive oxygen species, the damage to cell membrane integrity and cell wall after exposure to CILs.
It should however be noted that the oxidative stress theory of aging has to be further elucidated due to significant animal-strain-related differences in the effect of reactive oxygen species [ 61, 62].
This further supports that there are distinct differences in the formation of the reactive metabolite at lower methyl eugenol exposures.
MEU bioactivation and detoxification were studied by a physiologically based kinetic approach comparing human and rat models, and the results obtained revealed that there were no substantial species differences in the formation of the reactive ultimate carcinogenic l′-sulfoxymethyleugenol metabolite.
Although this classification is mainly based on histopathology, taking into account differences in the composition of the reactive infiltrate and stroma, recent studies have demonstrated that these disease entities are biologically different, with different genomic alterations, gene-expression patterns, cytokine milieu, and clinical behavior [ 6, 7].
In our cases, there were several clear differences between the reactive astrocytosis in the white and grey matter.
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