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However, there are differences in the linkage between fetal oxygenation and metabolic rate or growth for these cohort groups that may relate to underlying etiologic processes.
Zhu et al. (2003) did not observe any major differences in the linkage structure of the non-cellulosic polysaccharides in the CLSA9 defective mutant and hypothesized that the defect in binding A. tumefaciens may arise from the altered ability of the Arabidopsis mutant to secrete particular polysaccharides necessary for bacterial recognition of the host and subsequent attachment.
In Smith et al. (2011), we compare genome-wide LOD scores derived from SNPs genotyped from the Illumina TruSeq capture platform to those derived from a high-density SNP chip, showing very few differences in the linkage analysis results.
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Although CP5 and CP8 are comprised of identical monosaccharides (L-FucNAc, D-FucNAc and D-ManNAc), they are serologically distinct due to differences in the linkages between sugars and the sites of O-acetylation.
While it is possible that the two RNAs have very different ΔΔ G values for the states of SNF (which could account for the difference in the linkage effect), it is also possible that differences in the conformational landscapes of the RNAs (and how they bind protein) are important in the difference between αSLII and αSLIV.
The difference in the linkages between the oxalates and the Cd atoms in I III, produces unusual Cd O Cd one-dimensional chains, which have been observed for the first time.
However, differences in the plant linkage level were not significant; i.e. overall, plants were visited by similar numbers of insect species in invaded and non-invaded sites.
We hypothesize that the differences in stability between the AF488 and maytansine conjugates are related to the differences in the chemical linkages connecting the payload to the PEG2 portion of the linker (SI Figure S2).
Although we believe that this is a likely consequence of the differences in detailed linkage disequilibrium between the SNP markers and the actual pathogenic allelic variants at many of these chromosomal loci, some of these phase differences are also likely to reflect results that co-occur in the two independent samples by chance.
We observed little evidence of heterogeneity in allelic effects at these loci between groups of European and Japanese ancestry but obtained substantial improvements in the resolution of fine mapping of potential causal variants by leveraging transethnic differences in the distribution of linkage disequilibrium.
We also explored the possibility that failure to replicate individual SNPs could arise due to differences in the structure of linkage disequilibrium between discovery and replication subject groups as has been suggested by some investigators [11].
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