Sentence examples for differences in the hiv from inspiring English sources

From Table 2, we note that there were large differences in the HIV prevalence rates between the different ethnic groups.

In a reproducibility study, results obtained with the HRM assay were not significantly affected by differences in the HIV viral load of the plasma sample used for analysis (range: 2,000 to 50,000 copies/ml), the sample volume (100 vs. 500 ul), or the maximum number of HIV RNA copies used for amplification of DNA templates for HRM analysis (range 100 to 5,000 copies of HIV RNA).

There are no known, substantial differences in the HIV or TB epidemics across these 4 provinces.

Finally, local investigation of higher risk hospitals should be undertaken in order to disclose factors leading to differences in the HIV mortality risk detected in this study.

Early in the HIV epidemic, surveillance experts recognized that there were differences in the HIV epidemic between the urban and the rural areas [ 5, 6].

However, it is plausible that host population differences are related to the apparent selection differences, and further testing may reveal specific cause-and-effect relationships between host differences and rate differences in the HIV genome.

Although current analysis documents substantial differences in the utilisation of HIV care across Europe and Argentina, underlying reasons for these differences should be a subject for further investigation.

It is possible that, in some cases, the differences in the HIV-1 recovery levels observed in the different cultures as well as the failure of the drugs alone or in combination to stimulate HIV-1 in some cultures may be due to partioning of the few infected cells.

This variation between regions is possibly due to differences in the HIV-related burden of disease.

Furthermore HIV-1 specific CD8+ T cells persist in high numbers in persons with untreated chronic progressive disease and no quantitative differences in the HIV-1 specific T cell response were observed between individuals with progressive and long-term non-progressive infection [14] [16].

Based on this model and the lack of quantitative differences in the HIV-1 specific cellular immune response measured by IFN-γ secretion described above [14], [26], we hypothesized that HIV-1 Controllers would posses significantly higher numbers of terminally differentiated CD8+ T cells in directly isolated PBMC compared to HIV-1 Progressors.