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The differences in the critical clearance rate for different substitution therapies are minimized by keeping the fluctuations in the administered IgG dose per treatment small.
The two mechanisms have small differences in the critical initial Damkohler number.
In particular, significant differences in the critical shear required to stabilize turbulence are sometimes observed.
Most notably, this could include differences in the critical ion fluence needed to amorphize the two phases.
The differences in the "critical" concentration values (changing K1C from increasing to stabilization) for Al2O3 and NiO may be explained as effect of dopant type and difference in the methods of powder synthesis (co-precipitation and mixing).
Two alternative suggestions have been made to explain the observed differences in the critical stresses of the detwinning modes associated with the reverse and pseudo-reverse modes of deformation twinning.
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The compositional dependence of the susceptibility to the environmental embrittlement is considered to be due to the difference in the intrinsic grain boundary cohesion between two alloy compositions, i.e. to the difference in the critical hydrogen content causing intergranular cracking.
Neonates admitted to paediatric intensive care had significantly higher cTnT levels compared with the older intensive care admissions, but despite this there was no statistically significant difference in the critical illness score between these two subgroups.
Many systems of intra-provincial regionalization of care that are responsive to population density, geographic barriers, or evolved regional care systems also may lead to differences in the distribution of critical care services.
Little difference was observed in the critical values of 'Jingxiu', 'Riesling' and spine grape: all were approximately 47°C based on the Fv/Fm (from 46.5°C to 47.8°C) and Fo (from 46.5°C to 47.1°C) values.
Although it is difficult to reach a firm conclusion at this stage, our observations in the French family with 458dupA support the view that subtle differences in a critical region of the ferritin protein sequence modulate the phenotype of neuroferritinopathy.
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