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Existing differences can be explained to some extent by nonspecific answers of the patients and the absence of a clear definition for suicide attempts.
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Additionally, the study will reveal whether these differences can be explained by factors related to patients, nursing care or nursing home structure.
Probably, these differences can be explained by the bias inherent to each method, e.g. pyrosequencing and 16S rRNA gene library, used to survey the bacterial diversity.
These differences can be explained by different life history requirements relative to specific cover types.
The differences can be explained by both the methodologies used to derive the estimates and the greater number of surveys used to develop the model (534 additional surveys have become available since 2000).
Most of the differences can be explained by the use of different scales to measure PEF.
These differences can be explained by subjects' gain/loss utility relative to their region's reference point for trustworthiness.
These differences can be explained by the differences in the depth to bedrock and the soil stiffness profile between Korea and western US.
The clone to clone differences can be explained by unhomogenised rDNA pools containing multiple gene families and pseudogenised copies, as suggested by Galián et al. (2012).
These differences can be explained neither by changes in macrophage recruitment to vascular wall, which could explain the differences seen in antifibrotic phenotype, nor by different expression in MCP-1.
Such differences can be explained by using different ELISA kits with different cut off values.
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CEO of Professional Science Editing for Scientists @ prosciediting.com