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The stability of the mite populations, especially between generations, suggests to the authors that differences between the hosts' skin may explain why people from different regions have different subsets of the bugs.
Differences between the hosts appear with mutants of later steps in glycosylation and are, thus, likely to be due to different interactions between specifically glycosylated fungal proteins and their respective host receptors.
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The experimental results show that, there are significant differences between the host connectivity measured using TCP or ICMP; in general, the accuracy of connectivity measured using TCP is 20 30% higher than that measured using ICMP.
Therefore we chose to study this residue as its characteristics can shed light upon the differences between the host and the pathogen enzymes.
Moreover, by performing this screen in parallel with host cells derived from insects and humans we could readily assess the similarities and differences between the host factor dependencies.
While genetic differences between the host species are expected, it is not known what part, if any, genetic differentiation between the invading European and endemic Asian parasites plays.
The authors attributed better results with JM109 to the genetic differences between the host strains but did not delve deeper into the mechanisms underlying this phenotype.
We describe here the similarities and differences between the host transcriptional profiles of acute and convalescent KD subjects and profiles of children with acute bacterial and viral infections.
Therefore, the differences between the host factors underlying EHV-1 entry in equine and mouse cells may contribute to the difference in EHV-1 pathogenicity between the horse and the mouse model.
Seeking to identify differences between the host-responses to the two infected wound types, we mined more deeply into the dataset, analyzing all I/U-DEGs on POD4 with a p-value of ≤0.05 (Benjamini-Hochberg).
Similar to results from MβCD-treated HbAA parasitized erythrocytes, MβCD treatment collapsed the lifetime differences between the host cell membrane, PVM and PM of parasitized HbAS and HbSS cells (Fig. 5D,E).
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