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a At the first visit of the patient, giant polycystic liver, ascites, and elevated right diaphragm were observed.
Degenerative changes of the diaphragm were observed after keeping rats in hypercapnic chambers for 6 weeks or more [ 95, 96].
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In addition, minipores that are covered with a diaphragm are observed.
Selective angioocclusion, as controlled mainly by the optical field of exposure (diaphragm), was observed in the CAM chicken embryo model, following verteporfin therapy.
A large ovoid opening located in the left dorsolateral part of the diaphragm was observed (Fig. 1a, and b), affecting the muscular and the tendinous components.
In this area, the diaphragm is observed as a three-layered structure: a non-echogenic central layer bordered by two echogenic layers - the peritoneum and the diaphragmatic pleurae [ 27].
No visible diaphragm contractions were observed under mechanical ventilation.
No significant differences of fiber size variability were observed in the diaphragm and quadriceps (data not shown).
Other dystrophic alterations were observed in DMSXL diaphragm sections, such as centrally located nuclei, an increase in interfasicular connective tissue and the presence of infiltrating inflammatory cells (Fig. 2).
Crocidolite fibers were observed embedded in the diaphragm with activated mesothelial cells immediately after cessation of exposure.
In contrast, no metastases were observed in skeletal muscles of the limbs and diaphragm, which were examined in detail (Table 1).
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