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The result agrees satisfactorily with the available corresponding data for numerical solutions of the differential equations for the Higbie model, with the exception of one case, where the deviation is probably due to an error in the numerical solution, or its graphical representation.
The fact that this population showed no general heterozygote deficit suggests that deviation is probably due to the presence of null alleles.
Such a marked deviation is probably due to delayed transmission of drug information between the ward and the pharmacy, a situation that arises when parenteral antibiotics are prepared at the pharmacy and not near the patient.
Similar(57)
This difference is probably due to different funding systems [ 3].
This difference was probably due to the vernalization process.
One reason which might cause deviation from HWE is probably due to the loss of heterozygotes, which might be explained by the habitat fragmentation leading to further division of each population into small subpopulations causing an increase of homozygotes due to genetic drift and endogamy.
For that reason, we do not regard this deviation as being significant it is probably due to the combined effect of small statistics and large errors.
The relative lack of signal of population differentiation from RST is probably due to deviation from the assumed stepwise mutation model (Balloux and Lugon-Moulin 2002).
Rather, the inequality we find is probably due to slight deviations from dynamic equilibrium within an individual.
This is probably due a large standard deviation of the patients' responses with respect to controls (respectively, before ablation: cone a-wave SD 3.4 vs. 2.4 ms; cone b-wave SD 2.2 vs. 1.5 ms).
Moreover, the mean SUVr of DIPG were not significantly higher than those of the normal pons, but this is probably due to the small DIPG sample size as the standard deviations were high.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com