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Dysplasias are usually congenital abnormalities of tissue development or differentiation.
The development and function of ILCs are precisely regulated by a network of crucial transcription factors, which are also involved in the development or differentiation of conventional natural killer (cNK) cells and T cells.
This would suggest that this set of genes share a common mode of regulation during human development or differentiation.
However, there are no functional data addressing whether Id1 has a role in mammary development or differentiation in vivo.
Within our data set there are numerous examples of individual, differentially expressed genes that have not previously been implicated in inner ear development or differentiation.
Proteases can activate or truncate functions of proteins, unfold a cascade of events, trigger development or differentiation and cause cell death [1], [2].
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Transkingdom signaling is a mechanism in which molecules produced by bacteria, such as cyclodipeptides (CDPs), positively or negatively affect the development, growth, or differentiation of eukaryotic organisms.
In contrast, proteins that are required only for limited time in the cell, e.g. at a certain stage of the cell cycle, during development, growth or differentiation or in response to external stimuli, often have mRNAs with short half-lives.
Molecular markers can detect differences at the DNA level and offer numerous advantages over conventional phenotype-based alternatives because they are stable and detectable in all tissue types, regardless of the growth environment, development state, or differentiation status [ 27].
In many cases each of the alternative promoters is active in a particular cell type or at different stages of development or cell differentiation [35], [36], [41].
Early in mouse development or upon differentiation of embryonic stem (ES) cells, the XCI-activator concentration in a cell will increase, and in female cells this will drive the initiation of XCI with a specific probability.
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