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In the genome, the gene for Dali is situated next to a gene called Pou3f3, which encodes a protein that contributes to the growth and development of nerves and the kidneys.
The correlation of vasculature formation with the local development of nerves, mentioned above and elsewhere, called for modifying the concept of histo-hematoblasts to the organoblasts concept, to cover the observed phenomena more adequately.
These miRNAs are involved in cell differentiation [ 31, 32], and proliferation [ 33], and the development of nerves [ 34], heart [ 35], lung [ 36] and muscle [ 37, 38], suggesting that these miRNAs may play major roles in the regulation of fundamental biological processes, as well as the development of skin and HF.
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hnRNP A/B has been postulated to play important roles in differentiation of neural lineage and development of nerve system because of its high and broad expression in mouse developing brains and adult mature brains [ 118, 119].
Jaynes believed that the development of nerve fibres connecting the two hemispheres gradually integrated brain function.
Recent research suggests that it could be associated with the peregrinations of a single gene.Protocadherin is one of the proteins that guide the development of nerve cells.
The vitamin also appears to trigger the production of a protein called nerve growth factor, which directs the development of nerve cells and promotes their long-term survival.In this section Where's the smart money?
That these rhythmic contractions originate in the cardiac muscle can be substantiated by observing cardiac development in the embryo (see above); cardiac pulsations begin before adequate development of nerve fibres.
These data suggested that the BDNF/TrkB-mediated signaling pathway in the spinal cord was involved in the development of nerve injury-induced neuropathic pain through the activation of dorsal horn NMDA-2B receptors.
Thus, we examined the effect of the selective COX-1 inhibitor SC-560 [21] on the development of nerve injury-induced tactile allodynia.
We find here that neither antagonism of the MC1R nor a non-functional MC1R influences nerve-injury induced sensitisation, with all groups having a similar time course of development of nerve-injury induced sensitivity.
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