Sentence examples for development of goblet from inspiring English sources

By contrast, development of Goblet cells, enteroendocrine cells, and assembly of enterocyte apical junctions was not altered in hyh mutant mice.

Our results demonstrated that AR inhibition significantly prevented development of goblet cell metaplasia induced by IL-13 in human airway epithelial cells and by allergen (RWE) in mice.

These data suggest that smoking upregulates a systemic pattern of neutrophil and macrophage chemoattractant expression, and this correlates significantly with the development of goblet cell hyperplasia.

22,24,39 Previously, we have shown that the TH2-type immune response in resistance plays an important role in the development of goblet cell hyperplasia.

Staining of lung sections with PAS showed that immunisation with PBS (Fig. 3H) or gp140 (Fig. 3I) alone did not lead to the development of goblet cell metaplasia around the major airways.

It was also suggested that active signaling is present in the nasal mucosa of AR and that this may play a role in the development of goblet cell hyperplasia in AR [ 22].

Although there was significant impairment in the development of hyperplastic goblet cells in the KO mice, unexpectedly by day 15 after infection, there was an increase in PAS-positive goblet cells, with significant elevation by day 30 after infection.

Bi-allelic loss of Lkb1 led to the perturbed development of Paneth and goblet cell lineages.

Gestational SS exposure impairs the development of Clara and goblet cells but not ciliated epithelium cells.

These results indicate that prenatal exposure to SS impairs the development of Clara and goblet cells, which contributes to the reduced production of surfactant proteins and airway mucus in gestationally SS-exposed lungs.

In the present study, we show that gestational but not early postnatal exposure of mice to secondhand cigarette smoke (SS) suppresses alveolarization, angiogenesis, and development of Clara and goblet cells without significant lung inflammation and that the SS-induced effects on alveolar architecture are irreversible.