Cysts exposed for too long a period to water levels exceeding 65% will have completed their pre-emergence embryonic development; subsequent dehydration of these cysts will in the worst case result in the killing of the differentiated embryos.
Likely factors in the development of cerebral edema are dehydration, acidosis and low carbon dioxide levels; in addition, the increased level of inflammation and coagulation may, together with these factors, lead to decreased blood flow to parts of the brain, which then swells up once fluid replacement has been commenced.
Factors potentially implicated in the development of renal failure were dehydration (a history of several days' vomiting and/or diarrhea was reported in 75% of the cases), urinary tract infection (29%) and chronic renal dysfunction (21%).
Thus, while the physiological parameters at Tc,Max suggest that the animals had undergone heat-related dehydration, the development of tissue injury appeared to be a delayed process.
In addition, meprin A and filamin C will require further studies in patients with renal or urologic disease or extreme dehydration, and development of their clinical-grade assays will require normalization for potential variations in total urine protein concentration.
Single episodes of severe hyperglycemia, such as the in-hospital development of diabetic ketoacidosis or hyperglycemia-associated dehydration, may result in specific consequences, such as dialysis fistula thrombosis, readmission to a critical care unit, or treatment-related pulmonary edema.
In this series potential risk factors for the development of KD were pre-existing KD, dehydration, diabetes mellitus, shock, NSAIDs, aminoglycosides, ACE-I, AT-II, diuretics, heart failure, opiates, rhabdomyolysis and iodinated contrast agents.
Further analysis of five toxicity phenotypes in a stepwise logistic regression including only the c.−92A>G polymorphism, revealed an association between a heterozygous or homozygous c.−92A>G genotype and the development of grade 2 4 diarrhoea (P=0.002) and dehydration (P=0.042) in the first four cycles of chemotherapy (Table 8).
It is generally recognized that risk factors for the development of KD in this population include dehydration, increasing age, diabetes mellitus, shock, heart failure, medications (NSAIDs, aminoglycosides, ACE-I, diuretics, AT-II, opiates), rhabdomyolysis, iodinated contrast agents and pre-existing KD.
In the early stage of dehydration stress, the drought-tolerant cultivar might change their morphological characteristics to enhance root and leaf development, thus accumulating more biomass to counteract the wastage brought on by dehydration stress.
