Sentence examples for development of an ad from inspiring English sources

Going into the development of an ad with this thinking and testing was invaluable and saved time.

Interestingly, Elan and Wyeth used a similar strategy for the development of an AD protein vaccine (ACC-001), in which an N-terminal sequence of Aβ is conjugated to diphtheria toxin that should provide foreign Th epitope/s.

The development of an ad hoc scaffold for effective tissue engineering applications plays a pivotal role in promoting and support the healing process.

Because vaccinations may cause brain inflammation that leads to meningoencephalitis, minimizing the risk of such inflammation and meningoencephalitis has to be a major goal in the development of an AD vaccine.

Postnatal skin-specific inactivation of both RXRα and RXRβ (RXRs) leads to the development of an AD-like disease characterized by partial hair loss, Th2 inflammation, eosinophilia, elevated TSLP epidermal secretion, and high TSLP serum levels [24].

Histological analysis of the skin of control, RBP-JK5 and RBP-JK5/G-CSF+/− mice showed the development of an AD-like disease in both mutant mice, implying that reduced G-CSF serum levels did not ameliorate the skin phenotype (Figure 6C), which is expected as TSLP serum levels are still elevated in the RBPJ-K5/G-CSF+/− mice (Figure 6D).

Nevertheless, due to the lack of direct evidence of neurofibrillary tangle (NFT) deposition in the brain, we do not know whether hyperphosphorylated tau plays a role in promoting the development of an AD-like syndrome in experimental CT.

The funds will also be used to provide the vaccine and over 500 million auto-disable (AD) syringes and safety boxes to ensure safe injection practices and foster the development of a local AD syringe industry in China.

Here, we focus on the development of a CAT for the AD sub-scale, which contains 22 items.

The ablation of canonical Notch signaling in keratinocytes by simultaneous deletion of both Notch1 and Notch2 or RBP-J resulted in defective skin barrier function and increased expression of TSLP, leading to the development of a severe AD-like skin phenotype in mice (Demehri et al., 2008; Dumortier et al., 2010).

Although 'A β cascade theory' is one of the most accredited hypothesis to explain the development of AD, a growing body of evidence support a role of oxidative stress, as well as inflammation mechanisms in the pathogenesis of the disease.