Sentence examples for develop immunodeficiency from inspiring English sources

The role of Vpr in vivo has been investigated in rhesus macaques infected with SIVmac, and it was initially shown that monkeys infected with SIV lacking the vpr and the related vpx genes displayed a lower virus burden and did not consistently develop immunodeficiency disease [2], [3].

FIV infection causes gradually developing immunodeficiency and has only a minor impact on lifespan.

Natural hosts for simian immunodeficiency virus (SIV) can be, and are often naturally, infected with species-specific SIVs, but do not develop acquired immunodeficiency syndrome (AIDS).

FIV infection of cats provides an important animal model to address this significant question because cats are the only hosts that develop an immunodeficiency syndrome from a natural lentivirus infection that can be used for experimental studies.

Hence, starting ART very early reduces disease progression and early mortality; close follow up of all children of HIV-positive mothers is recommended to make the diagnosis and start treatment at an earlier time before they develop severe immunodeficiency.

Françoise naturally focussed on human immunodeficiency virus (HIV) and acquired immune deficiency syndrome (AIDS) and the insights that studies on human HIV controllers and primate models that do not develop simian immunodeficiency virus (SIV) disease have brought to our understanding of HIV pathogenesis and protection from disease.

2 Previous research suggests that a person's SES may affect his or her likelihood of contracting HIV and developing acquired immunodeficiency syndrome (AIDS).

This may be explained by the fact that HIV-infected patients live longer and are at higher risk of developing nonacquired immunodeficiency syndrome (AIDS) comorbid illnesses, as well as problems related to immune reconstitution inflammatory response syndrome and toxicities related to cART [ 4, 7- 9].

Not surprisingly, this did not ultimately inhibit the development of SIV-specific T or B cell responses, since these typically develop in most immunodeficiency virus infected animals.

Furthermore, our patients did not develop any humoral immunodeficiency related to repeated treatment [ 50].

Nonetheless, SM1 and SM2 did not develop signs of clinical immunodeficiency throughout 13 years of follow-up.