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Firstly, clinical data showed that the index patient did not develop diarrhoea during course of disease.
He did not develop diarrhoea during the course of the disease.
They show that LPS-treated mice develop diarrhoea, which correlates with intestinal epithelial cell shedding and caspase-3-driven apoptosis.
Nevertheless, in the multivariate analysis, patients with UGT1A7*3/*3 and UGT1A9*1/*1 genotypes were more likely to develop diarrhoea after first cycle of chemotherapy (Table 4).
All participants who develop diarrhoea during the study period will be asked to provide a stool sample and this will be collected during a home visit if required.
Likewise, the former group were more likely to develop diarrhoea, 38% (21/54) versus 21% (3/14), with three patients positive for Clostridium difficile toxin in the former group (none in the latter).
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And he developed diarrhoea of the mouth.
In the early days of antibiotics, patients frequently developed diarrhoea from the massive killing-off of normal bacteria.
13.3%, 13.2%, and 10.3% of children in the SP plus AQ, DHA plus PQ and SP plus PQ groups respectively developed diarrhoea, compared to 17.5% in the control group.
On day 56, the patient developed diarrhoea, both pp65Ag and plasma PCR were negative.
Exclusive breast feeding for 6 months was protective against developing diarrhoea.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com