Sentence examples for determining this phenomenon from inspiring English sources

The hormonal pathways determining this phenomenon are still unknown.

In an experimental model of cardiac arrest and resuscitation, Tang et al. [ 16] demonstrated that post-resuscitation myocardial dysfunction was more severe in animals treated with epinephrine than in those treated with the α-agonist, phenylephrine, or with a combination of epinephrine and the β1-blocker, esmolol, thus suggesting that β1 stimulation has a role in determining this phenomenon.

Previously, we have demonstrated that vesicles from the apical cell surface are shed into urine because of tip vesiculation of microvilli, with u-PCX reflecting the urinary shedding of microvilli; we also determined this phenomenon to be common in glomerular diseases [ 8].

Further study will be necessary to determine if this phenomenon can significantly influence extinction probabilities.

This raises the prospect that the observed uptake of [18F]FDG-6-P inthethe other organs may be a real process and so warrants further investigation to determine whether this phenomenon extends to man.

To determine whether this phenomenon occurs in opioid-dependent individuals, eight sevenn male, one female) methadone-maintained cocaine smokers participated in a 47-day inpatient and outpatient study to assess the effects of memantine on smoked cocaine self-administration, subjective effects, and cardiovascular responses.

Although it still must be determined if this phenomenon occurs in humans, these results may have far reaching implications.

To determine whether this phenomenon was also happening in vivo, we stained normal mammary tissues from VDR WT and VDR KO mice for TCF-4.

To determine if this phenomenon was cell autonomous and mediated by a loss of torsinA function in Purkinje cells, we created a knockout of the Dyt1 gene only in Purkinje cells of mice.

This work raises interesting questions about whether A. tumefaciens may be able to transform organisms other than plants in nature, or indeed should be considered during GM risk assessments, with further investigations required to determine whether this phenomenon has already occurred in nature.

Since it has been reported that the ΔGAG mutation causes a reduction of torsinA in the striatum and the entire brain [26], [27], [28], we sought to determine if this phenomenon was mediated by a loss of function of torsinA in Purkinje cells by creating a knockout of the Dyt1 gene only in Purkinje cells (Dyt1 pKO) of mice.