While larger and better-controlled clinical studies are needed to determine this finding's significance – for example, whether altered monocyte subset numbers portend prognosis in patients with atherosclerosis – this preliminary enumeration reveals a sufficient sensitivity of a DMR-chip assay to detect values observed in the clinic.
We now (1) determined whether this finding could be replicated in a new sample of cases and controls, and (2) analyzed HA sub-scores (HA1−HA4) to further understand case control differences.
It remains to be determined whether this finding merely reflects the high number of domestically obtained infections and closer travel destinations or supports the hypothesis that highly susceptible isolates might cause a more severe disease, such as bacteremia.
Afterwards, an expression of the splay state is determined: this task corresponds to finding a fixed point of the event-driven map in a suitably moving reference frame - analogously to what has previously been done in specific contexts [21, 25, 26].
Further study is needed to determine whether this finding is explained by left turns across traffic exposing right-seated passengers to same-side crashes from faster moving oncoming traffic compared to left-seated passengers who may be less frequently exposed to high-speed turn-related crashes.
To determine if this finding would further apply to neoplasia, we evaluated Metnase and Topo IIα expression in four breast cell lines.
Given that PrECs showed higher levels of phospho-ELK than any of the prostate cancer cell lines, we wanted to determine if this finding was in harmony with phospho-ELK levels in tumoral and normal prostate tissues.
Ongoing injury surveillance is required to determine if this finding is repeated in future tournaments.
More isolates are needed to determine whether this finding is indicative of an expanding clone.
However, a larger study would be needed to determine whether this finding is consistent across other practices.
