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T2D was diagnosed on clinically determined absence of type 1 or secondary diabetes, in individuals of more than 40 years of age at onset and without insulin treatment within 2 years after disease onset.
When BMI was not indicative of the presence of malnutrition (>18.5 or >21 before or after 70 years of age, respectively) and WL could not be determined (absence of weight data 6 months earlier), we considered that malnutrition could not be eliminated and patients were not analysed for the association with risk factors or outcomes.
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They found that the simplified disease activity index was superior to the DAS28 in determining absence of inflammatory activity, and therefore in determining remission [ 17].
However, they are unable to multiply (in vitro or in vivo) and establish an infection as determined by absence of parasitemia.
We then determined whether absence of contractile activity affects α-actinin transcription.
Progression-free survival (PFS) was determined as absence of local or distant recurrence/progression or death of any cause.
Moreover, we determined the absence of the 2-oxoacid-dependend oxygenase activity GSL-OH in Aethionema (table 3).
RT PCR and western blot analyses determined the absence of TBPH RNA and protein (Fig. 2B and C).
Peripheral ischemia was determined by absence of both dorsalis pedis and posterior tibial pulses on clinical palpation.
We next determined whether absence of the ST2-mediated signalling pathway might protect in another model of colitis that is induced by DSS.
All samples were tested in triplicate, and the coefficient of variation (CV) was calculated in the triplicates of cycle threshold (CT) to determine the absence of technical and manipulation errors.
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