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Our new findings also expand our previous report describing deficient VEP habituation at rest in migraine patients between attacks [21, 22] now showing that HV worsens this deficit mainly by decreasing further the already abnormally low amplitude in the first VEP block.
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For example investigating evoked and event-related potentials, Gerber and Schoenen et al. [39, 40] described a deficient habituation, or even potentiation, of cortical responses during repetitive stimulation between attacks in patients suffering from migraine.
Several recent papers also described Th1 deficient functions of circulating myeloid DC in specific conditions depending on the activation pathway of DC [8], [16], [53].
Using our recently described var2csa deficient parasite line FCR3Δvar2csa 1F1 [10], we were able to evaluate the role of non-CSA placental adhesion receptors.
Motivation: Many published manuscripts contain experiment protocols which are poorly described or deficient in information.
However, many, perhaps most, scientific protocols in the literature are so poorly described and deficient in information that their exact repetition is impossible.
Thus, the sequencing of 16S rDNA can serve as a supplement in regular clinical work for bacteria identification, especially for poorly described, biochemically deficient or fastidious organisms.
Moreover, while increased ROS production was a feature in NDUFS4 deficient patient and mouse fibroblasts [ 17, 46], there was little evidence of this in our Ndufs4 fky/fky primary astrocytes, nor the previously described NDUFS4 deficient mesencephalic neurons [ 20, 21].
The BBDR HPT axis model parameters had been calibrated for the euthyroid adult rat previously and used to describe iodide-deficient conditions (McLanahan et al. 2008).
Experiments performed on this patient's fibroblasts yielded similar defects albeit less severe as found in the cells of the previously described COG-deficient patients.
In addition to the previously described SDH-deficient, IGF1Rhigh wild-type GISTs, other SDH-intact wild-type subpopulations were defined by high relative expression of IGF1R, neural markers, IGF1 and INSR, or low IGF1R coupled with high IGF2.
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