We assessed the reliability of P i,j x, y by the 5% significance Kolmogorov Smirnov test to examine if the values derived from the replications followed a Gaussian distribution and by the 15% bootstrap percentile method to test if the values obtained using the entire data set were within a 15% deviation of the average values calculated from the replication.
This section aims to provide more concrete details about various 'architecture work' derived from the 'data replication service' use case guided by the reference model introduced in Section "Data infrastructure reference model" as frame of reference.
The results showed that a low level of luciferase mRNA was generated either by cryptic promoters or by splicing of cryptic introns within the replicon transcripts in nucleus but not in the mRNAs derived from the cytoplasmic replication and transcription of the replicon (data not shown).
1- Data derived from the average replication time from two synchronizations.
The retroviral restriction factor TRIMCyp, which is a fusion protein derived from the TRIM5 gene, blocks replication at a post-entry step.
The accuracy of DNA replication is derived from the balance between three important components: base selectivity by the replicative DNA polymerases (pols), exonucleolytic proofreading, and post-replicative mismatch repair.
We found that the copy number of mtDNA in R2 and R3 samples of RH1 dsRNA-treated cells became significantly lower than that in D3 samples, yet the mtDNA replication intermediates derived from the strand-coupled DNA synthesis were relatively easily detected in the R2 sample without S1 nuclease treatment.
Two synthetic peptides, derived from the relevant gp120 sequence inhibited HIV-1 replication in macrophages and T lymphocytes in sequence-dependent manner.
We then found that ε = 127,665 could provide a reasonable description of genome replication in the dark, if all dNTPs used in phage genome replication in the dark were derived from the host genome (Fig. 3).
Double-stranded RNA (dsRNA) is the factor triggering the process and upon infection the host silencing machinery is induced by dsRNAs generated during viral replication or secondary structures derived from the pathogen genome [ 5- 7].
Primarily, this was an investigation of a single disease (schizophrenia) where the SNPs derived from the discovery sample were tested in an independent schizophrenia replication sample.
