Sentence examples for dependence on the cellular from inspiring English sources

Therefore, HCV was ultraviolet inactivated before infection indicating a dependence on the cellular recognition of HCV replication products [ 50 ].

However, our results imply that in the development of therapeutic strategies for blocking Stat3 in breast cancer cells, the strong dependence on the cellular context that this factor activity displays should be taken into account.

The dependence of the cellular macromolecular composition on growth rate has been a focus of intense investigation for over 30 years [ 45, 46].

However, the area of the Z-ring that is occupied by FtsZ-mEos2 molecules, showed a linear dependence on the total cellular level of FtsZ (Figure 7B).

It is generally assumed that DNA viruses evolve at a similar rate to that of their hosts due to viral dependence on the host's cellular machinery for replication [44], [45].

The use of, or even the dependence on, certain cellular factors by HIV-1 can differ between the various target cells such as T cells and macrophages.

These studies further imply that T3 may foster hepatic homeostasis by controlling the growth of either adult hepatocytes or hepatic neoplasm; however, the various reported effects of TH on cellular proliferation suggest a dependence on the specific cell type and cellular microenvironment.

Moreover, the dependence of induced gene expression on the cellular abundance of the transactivator provides important evidence to help explain the large difference of effector concentrations reported to fully activate TRPs in various cell systems.

Previous studies have shown that Wnt/β-catenin signaling participates in EMT in numerous cancers; however, the phenotypes and downstream molecular events are fairly different, reflecting the dependence on cellular context and tissue specificity [ 39].

Cancer cell heterogeneity and the continued genomic diversity acquired from one cancer cell division to another may promote cancer cell stress or dependence on alternative cellular pathways that are potentially targetable, as witnessed by success with poly(ADP-ribose) polymerase inhibition in patients who harbour germline BRCA1/2 mutations [ 7, 8].

Simply put, NFV enhances cellular dependence on the proteasome, which could be exploited for cancer therapy.